Robert Kleemann

Atherosclerosis and liver inflammation induced by increased dietary cholesterol intake: a combined transcriptomics and metabolomics analysis (2007)

Kleemann, Robert, Verschuren, Lars, Van Erk, Marjan J, Nikolsky, Yuri, Cnubben, Nicole HP, Verheij, Elwin R, ...

Abstract Background Increased dietary cholesterol intake is associated with atherosclerosis. Atherosclerosis development requires a lipid and an inflammatory component. It is unclear where and how...

MIF is a noncognate ligand of CXC chemokine receptors in inflammatory and atherogenic cell recruitment (2007)

Bernhagen, Jurgen, Krohn, Regina, Lue, Hongqi, Gregory, J. L., Zernecke, Alma, Koenen, Rory R., ...

© 2007 Nature Publishing Group

Über das Endresultat operierter Ellenbogengelenk-Tuberkulosen : Aus d. Chir. Klinik zu Kiel. (1916)

Kleemann, Robert.

Kiel, Med. Diss., 1916.

Functional Characterization of the Plasmodium falciparum and P. berghei Homologues of Macrophage Migration Inhibitory Factor▿

Augustijn, Kevin D., Kleemann, Robert, Thompson, Joanne, Kooistra, Teake, Crawford, Carina E., Reece, Sarah E., ...

Macrophage migration inhibitory factor (MIF) is a mammalian cytokine that participates in innate and adaptive immune responses. Homologues of mammalian MIF have been discovered in parasite species...

Collagen Degradation in the Abdominal Aneurysm : A Conspiracy of Matrix Metalloproteinase and Cysteine Collagenases

Abdul-Hussien, Hazem, Soekhoe, Ratna G.V., Weber, Ekkehard, Von Der Thüsen, Jan H., Kleemann, Robert, Mulder, Adri, ...

Growth and rupture of abdominal aortic aneurysms (AAAs) result from increased collagen turnover. Collagen turnover critically depends on specific collagenases that cleave the triple helical region of...

Atherosclerosis and liver inflammation induced by increased dietary cholesterol intake: a combined transcriptomics and metabolomics analysis

Kleemann, Robert, Verschuren, Lars, Van Erk, Marjan J, Nikolsky, Yuri, Cnubben, Nicole HP, Verheij, Elwin R, ...

With increasing dietary cholesterol intake the liver switches from a mainly resilient to a predominantly inflammatory state, which is associated with early lesion formation.