Lutz Schomburg

Hepatically derived selenoprotein P is a key factor for kidney but not for brain selenium supply

Schweizer, Ulrich, Streckfuß, Florian, Pelt, Paco, Carlson, Bradley A., Hatfield, Dolph L., Köhrle, Josef, ...

Liver-specific inactivation of Trsp, the gene for selenocysteine tRNA, removes SePP (selenoprotein P) from plasma, causing serum selenium levels to fall from 298 μg/l to 50 μg/l and kidney...

Gene disruption discloses role of selenoprotein P in selenium delivery to target tissues.

Schomburg, Lutz, Schweizer, Ulrich, Holtmann, Bettina, Flohé, Leopold, Sendtner, Michael, Köhrle, Josef

Selenoprotein P (SePP), the major selenoprotein in plasma, has been implicated in selenium transport, selenium detoxification or antioxidant defence. We generated SePP-knockout mice that were viable,...

Efficient selenium transfer from mother to offspring in selenoprotein-P-deficient mice enables dose-dependent rescue of phenotypes associated with selenium deficiency.

Schweizer, Ulrich, Michaelis, Marten, Köhrle, Josef, Schomburg, Lutz

Mice deficient in selenoprotein P exhibit a disturbed selenium distribution and reduced activities of other selenoenzymes and display defects in growth and motor co-ordination. We have normalized...

Hepatically derived selenoprotein P is a key factor for kidney but not for brain selenium supply

Schweizer, Ulrich, Streckfuß, Florian, Pelt, Paco, Carlson, Bradley A., Hatfield, Dolph L., Köhrle, Josef, ...

Liver-specific inactivation of Trsp, the gene for selenocysteine tRNA, removes SePP (selenoprotein P) from plasma, causing serum selenium levels to fall from 298 μg/l to 50 μg/l and kidney...

Gene disruption discloses role of selenoprotein P in selenium delivery to target tissues.

Schomburg, Lutz, Schweizer, Ulrich, Holtmann, Bettina, Flohé, Leopold, Sendtner, Michael, Köhrle, Josef

Selenoprotein P (SePP), the major selenoprotein in plasma, has been implicated in selenium transport, selenium detoxification or antioxidant defence. We generated SePP-knockout mice that were viable,...

Efficient selenium transfer from mother to offspring in selenoprotein-P-deficient mice enables dose-dependent rescue of phenotypes associated with selenium deficiency.

Schweizer, Ulrich, Michaelis, Marten, Köhrle, Josef, Schomburg, Lutz

Mice deficient in selenoprotein P exhibit a disturbed selenium distribution and reduced activities of other selenoenzymes and display defects in growth and motor co-ordination. We have normalized...