Joseph P. Grande

Publication List Details

Period

2007 - 2007

Number

8

Co-Authors

Effects of high-fat diet and/or body weight on mammary tumor leptin and apoptosis signaling pathways in MMTV-TGF-α mice (2007)

Dogan, Soner, Hu, Xin, Zhang, Yan, Maihle, Nita J, Grande, Joseph P, Cleary, Margot P

Abstract Introduction Obesity is a risk factor for postmenopausal breast cancer and is associated with shortened mammary tumor (MT) latency in MMTV-TGF-α mice with dietary-induced obesity. One link...

Transgenic Sickle Mice Are Markedly Sensitive to Renal Ischemia-Reperfusion Injury

Nath, Karl A., Grande, Joseph P., Croatt, Anthony J., Frank, Elena, Caplice, Noel M., Hebbel, Robert P., ...

Ischemic injury is invoked as a mechanism contributing to end-organ damage and other complications of sickle cell disease (SCD). However, the intrinsic sensitivity of tissues in SCD to ischemic...

Anomalous Renal Effects of Tin Protoporphyrin in a Murine Model of Sickle Cell Disease

Juncos, Julio P., Grande, Joseph P., Murali, Narayana, Croatt, Anthony J., Juncos, Luis A., Hebbel, Robert P., ...

In human and murine models of sickle cell disease (SCD), heme oxygenase-1 (HO-1) is induced in the kidney, an organ commonly involved in SCD. The present study assessed the role of HO-1 by using a...

The Indispensability of Heme Oxygenase-1 in Protecting against Acute Heme Protein-Induced Toxicity in Vivo

Nath, Karl A., Haggard, Jill J., Croatt, Anthony J., Grande, Joseph P., Poss, Kenneth D., Alam, Jawed

Heme oxygenase (HO) is the rate limiting enzyme in the degradation of heme, and its isozyme, HO-1, may protect against tissue injury. One posited mechanism is the degradation of heme released from...

Oxidative Stress and Induction of Heme Oxygenase-1 in the Kidney in Sickle Cell Disease

Nath, Karl A., Grande, Joseph P., Haggard, Jill J., Croatt, Anthony J., Katusic, Zvonimir S., Solovey, Anna, ...

Chronic nephropathy is a recognized complication of sickle cell disease. Using a transgenic sickle mouse, we examined whether oxidative stress occurs in the sickle kidney, the origins and functional...

Increased Venous Proinflammatory Gene Expression and Intimal Hyperplasia in an Aorto-Caval Fistula Model in the Rat

Nath, Karl A., Kanakiriya, Sharan K. R., Grande, Joseph P., Croatt, Anthony J., Katusic, Zvonimir S.

We hypothesized that the venous limb of an arteriovenous (AV) fistula would evince up-regulation of genes relevant to vascular remodeling along with neointimal hyperplasia and relevant histological...

Renal Hemodynamic, Inflammatory, and Apoptotic Responses to Lipopolysaccharide in HO-1−/− Mice

Tracz, Michal J., Juncos, Julio P., Grande, Joseph P., Croatt, Anthony J., Ackerman, Allan W., Rajagopalan, Govindarajan, ...

Lipopolysaccharide (LPS) induces the stress-responsive gene heme oxygenase-1 (HO-1). The present study examined the significance of HO-1 in response to LPS. In HO-1−/− mice, as compared with...