Jeffrey D. Ceci

Tumor induction by an Lck-MyrAkt transgene is delayed by mechanisms controlling the size of the thymus

Malstrom, Scott, Tili, Esmerina, Kappes, Dietmar, Ceci, Jeffrey D., Tsichlis, Philip N.

Transgenic mice expressing MyrAkt from a proximal Lck promoter construct develop thymomas at an early age, whereas transgenic mice expressing constitutively active Lck-AktE40K develop primarily...

The metabolic syndrome resulting from a knockout of the NEIL1 DNA glycosylase

Vartanian, Vladimir, Lowell, Brian, Minko, Irina G., Wood, Thomas G., Ceci, Jeffrey D., George, Shakeeta, ...

Endogenously formed reactive oxygen species continuously damage cellular constituents including DNA. These challenges, coupled with exogenous exposure to agents that generate reactive oxygen species,...

Tumor induction by an Lck-MyrAkt transgene is delayed by mechanisms controlling the size of the thymus

Malstrom, Scott, Tili, Esmerina, Kappes, Dietmar, Ceci, Jeffrey D., Tsichlis, Philip N.

Transgenic mice expressing MyrAkt from a proximal Lck promoter construct develop thymomas at an early age, whereas transgenic mice expressing constitutively active Lck-AktE40K develop primarily...

The metabolic syndrome resulting from a knockout of the NEIL1 DNA glycosylase

Vartanian, Vladimir, Lowell, Brian, Minko, Irina G., Wood, Thomas G., Ceci, Jeffrey D., George, Shakeeta, ...

Endogenously formed reactive oxygen species continuously damage cellular constituents including DNA. These challenges, coupled with exogenous exposure to agents that generate reactive oxygen species,...