Hein te Riele

Restriction beyond the restriction point: mitogen requirement for G2passage (2006)

Foijer, Floris, Te Riele, Hein

Abstract Cell proliferation is dependent on mitogenic signalling. When absent, normal cells cannot pass the G 1 restriction point, resulting in cell cycle arrest. Passage through the G 1 restriction...

Mouse embryonic stem cells carrying one or two defective Msh2 alleles respond abnormally to oxidative stress inflicted by low-level radiation

DeWeese, Theodore L., Shipman, Jennifer M., Larrier, Nicole A., Buckley, Nicola M., Kidd, LaCreis R., Groopman, John D., ...

Chronic oxidative stress may play a critical role in the pathogenesis of many human cancers. Here, we report that mouse embryonic stem (ES) cells deficient in DNA mismatch repair responded abnormally...

Msh2 status modulates both apoptosis and mutation frequency in the murine small intestine

Toft, Neil J., Winton, Douglas J., Kelly, Jane, Howard, Louise A., Dekker, Marleen, Te Riele, Hein, ...

Deficiency in genes involved in DNA mismatch repair increases susceptibility to cancer, particularly of the colorectal epithelium. Using Msh2 null mice, we demonstrate that this genetic defect...

Overlapping Functions of the pRb Family in the Regulation of rRNA Synthesis

Ciarmatori, Sonia, Scott, Pamela H., Sutcliffe, Josephine E., McLees, Angela, Alzuherri, Hadi M., Dannenberg, Jan-Hermen, ...

The “pocket” proteins pRb, p107, and p130 are a family of negative growth regulators. Previous studies have demonstrated that overexpression of pRb can repress transcription by RNA polymerase...

E2F mediates cell cycle-dependent transcriptional repression in vivo by recruitment of an HDAC1/mSin3B corepressor complex

Rayman, Joseph B., Takahashi, Yasuhiko, Indjeian, Vahan B., Dannenberg, Jan-Hermen, Catchpole, Steven, Watson, Roger J., ...

Despite biochemical and genetic data suggesting that E2F and pRB (pocket protein) families regulate transcription via chromatin-modifying factors, the precise mechanisms underlying gene regulation by...

Targeted gene modification in mismatch-repair-deficient embryonic stem cells by single-stranded DNA oligonucleotides

Dekker, Marleen, Brouwers, Conny, Te Riele, Hein

Gene targeting through homologous recombination in murine embryonic stem (ES) cells is already strongly suppressed by DNA mismatch-repair (MMR)-dependent anti-recombination when targeting construct...

CTG repeat instability and size variation timing in DNA repair-deficient mice

Savouret, Cédric, Brisson, Edith, Essers, Jeroen, Kanaar, Roland, Pastink, Albert, Te Riele, Hein, ...

Type 1 myotonic dystrophy is caused by the expansion of an unstable CTG repeat in the DMPK gene. We have investigated the molecular mechanisms underlying the CTG repeat instability by crossing...

Mouse MutS-like protein Msh5 is required for proper chromosome synapsis in male and female meiosis

De Vries, Sandra S., Baart, Esther B., Dekker, Marleen, Siezen, Ariaan, De Rooij, Dirk G., De Boer, Peter, ...

Members of the mammalian mismatch repair protein family of MutS and MutL homologs have been implicated in postreplicative mismatch correction and chromosome interactions during meiotic recombination....

p107 is a suppressor of retinoblastoma development in pRb-deficient mice

Robanus-Maandag, Els, Dekker, Marleen, Van Der Valk, Martin, Carrozza, Maria-Luisa, Jeanny, Jean-Claude, Dannenberg, Jan-Hermen, ...

Hemizygosity for the retinoblastoma gene RB in man strongly predisposes to retinoblastoma. In the mouse, however, Rb hemizygosity leaves the retina normal, whereas in Rb−/− chimeras pRb-deficient...

Ablation of the Retinoblastoma gene family deregulates G1 control causing immortalization and increased cell turnover under growth-restricting conditions

Dannenberg, Jan-Hermen, Van Rossum, Agnes, Schuijff, Leontine, Te Riele, Hein

The retinoblastoma suppressor pRB belongs to the family of so-called pocket proteins, which also includes p107 and p130. These proteins may functionally overlap in cell cycle control and tumor...

MSH2-Dependent Germinal CTG Repeat Expansions Are Produced Continuously in Spermatogonia from DM1 Transgenic Mice

Savouret, Cédric, Garcia-Cordier, Corinne, Megret, Jérôme, Te Riele, Hein, Junien, Claudine, Gourdon, Geneviève

Myotonic dystrophy type 1 is a neuromuscular affection associated with the expansion of an unstable CTG repeat in the DM protein kinase gene. The disease is characterized by somatic tissue-specific...

Pocket Protein Complexes Are Recruited to Distinct Targets in Quiescent and Proliferating Cells

Balciunaite, Egle, Spektor, Alexander, Lents, Nathan H., Cam, Hugh, Te Riele, Hein, Scime, Anthony, ...

Biochemical and genetic studies have determined that retinoblastoma protein (pRB) tumor suppressor family members have overlapping functions. However, these studies have largely failed to distinguish...

Restriction beyond the restriction point: mitogen requirement for G2 passage

Foijer, Floris, Te Riele, Hein

Cell proliferation is dependent on mitogenic signalling. When absent, normal cells cannot pass the G1 restriction point, resulting in cell cycle arrest. Passage through the G1 restriction point...

Mouse embryonic stem cells carrying one or two defective Msh2 alleles respond abnormally to oxidative stress inflicted by low-level radiation

DeWeese, Theodore L., Shipman, Jennifer M., Larrier, Nicole A., Buckley, Nicola M., Kidd, LaCreis R., Groopman, John D., ...

Chronic oxidative stress may play a critical role in the pathogenesis of many human cancers. Here, we report that mouse embryonic stem (ES) cells deficient in DNA mismatch repair responded abnormally...

Msh2 status modulates both apoptosis and mutation frequency in the murine small intestine

Toft, Neil J., Winton, Douglas J., Kelly, Jane, Howard, Louise A., Dekker, Marleen, Te Riele, Hein, ...

Deficiency in genes involved in DNA mismatch repair increases susceptibility to cancer, particularly of the colorectal epithelium. Using Msh2 null mice, we demonstrate that this genetic defect...

Overlapping Functions of the pRb Family in the Regulation of rRNA Synthesis

Ciarmatori, Sonia, Scott, Pamela H., Sutcliffe, Josephine E., McLees, Angela, Alzuherri, Hadi M., Dannenberg, Jan-Hermen, ...

The “pocket” proteins pRb, p107, and p130 are a family of negative growth regulators. Previous studies have demonstrated that overexpression of pRb can repress transcription by RNA polymerase...

E2F mediates cell cycle-dependent transcriptional repression in vivo by recruitment of an HDAC1/mSin3B corepressor complex

Rayman, Joseph B., Takahashi, Yasuhiko, Indjeian, Vahan B., Dannenberg, Jan-Hermen, Catchpole, Steven, Watson, Roger J., ...

Despite biochemical and genetic data suggesting that E2F and pRB (pocket protein) families regulate transcription via chromatin-modifying factors, the precise mechanisms underlying gene regulation by...

Targeted gene modification in mismatch-repair-deficient embryonic stem cells by single-stranded DNA oligonucleotides

Dekker, Marleen, Brouwers, Conny, Te Riele, Hein

Gene targeting through homologous recombination in murine embryonic stem (ES) cells is already strongly suppressed by DNA mismatch-repair (MMR)-dependent anti-recombination when targeting construct...

CTG repeat instability and size variation timing in DNA repair-deficient mice

Savouret, Cédric, Brisson, Edith, Essers, Jeroen, Kanaar, Roland, Pastink, Albert, Te Riele, Hein, ...

Type 1 myotonic dystrophy is caused by the expansion of an unstable CTG repeat in the DMPK gene. We have investigated the molecular mechanisms underlying the CTG repeat instability by crossing...

Mouse MutS-like protein Msh5 is required for proper chromosome synapsis in male and female meiosis

De Vries, Sandra S., Baart, Esther B., Dekker, Marleen, Siezen, Ariaan, De Rooij, Dirk G., De Boer, Peter, ...

Members of the mammalian mismatch repair protein family of MutS and MutL homologs have been implicated in postreplicative mismatch correction and chromosome interactions during meiotic recombination....

p107 is a suppressor of retinoblastoma development in pRb-deficient mice

Robanus-Maandag, Els, Dekker, Marleen, Van Der Valk, Martin, Carrozza, Maria-Luisa, Jeanny, Jean-Claude, Dannenberg, Jan-Hermen, ...

Hemizygosity for the retinoblastoma gene RB in man strongly predisposes to retinoblastoma. In the mouse, however, Rb hemizygosity leaves the retina normal, whereas in Rb−/− chimeras pRb-deficient...

Ablation of the Retinoblastoma gene family deregulates G1 control causing immortalization and increased cell turnover under growth-restricting conditions

Dannenberg, Jan-Hermen, Van Rossum, Agnes, Schuijff, Leontine, Te Riele, Hein

The retinoblastoma suppressor pRB belongs to the family of so-called pocket proteins, which also includes p107 and p130. These proteins may functionally overlap in cell cycle control and tumor...

MSH2-Dependent Germinal CTG Repeat Expansions Are Produced Continuously in Spermatogonia from DM1 Transgenic Mice

Savouret, Cédric, Garcia-Cordier, Corinne, Megret, Jérôme, Te Riele, Hein, Junien, Claudine, Gourdon, Geneviève

Myotonic dystrophy type 1 is a neuromuscular affection associated with the expansion of an unstable CTG repeat in the DM protein kinase gene. The disease is characterized by somatic tissue-specific...

Pocket Protein Complexes Are Recruited to Distinct Targets in Quiescent and Proliferating Cells

Balciunaite, Egle, Spektor, Alexander, Lents, Nathan H., Cam, Hugh, Te Riele, Hein, Scime, Anthony, ...

Biochemical and genetic studies have determined that retinoblastoma protein (pRB) tumor suppressor family members have overlapping functions. However, these studies have largely failed to distinguish...

Restriction beyond the restriction point: mitogen requirement for G2 passage

Foijer, Floris, Te Riele, Hein

Cell proliferation is dependent on mitogenic signalling. When absent, normal cells cannot pass the G1 restriction point, resulting in cell cycle arrest. Passage through the G1 restriction point...

Generation of a mouse mutant by oligonucleotide-mediated gene modification in ES cells

Aarts, Marieke, Dekker, Marleen, De Vries, Sandra, Van Der Wal, Anja, Te Riele, Hein

Oligonucleotide-mediated gene targeting is emerging as a powerful tool for the introduction of subtle gene modifications in mouse embryonic stem (ES) cells and the generation of mutant mice. However,...